Last edited by Tenris
Sunday, February 2, 2020 | History

4 edition of Gene regulation and fetal development found in the catalog.

Gene regulation and fetal development

proceedings of the Third International Workshop on Fetal Genetic Pathology, June 3-6, 1993

by International Workshop on Fetal Genetic Pathology (3rd 1993 Perugia, Italy)

  • 379 Want to read
  • 7 Currently reading

Published by Wiley-Liss in New York .
Written in English

    Subjects:
  • Fetus -- Abnormalities -- Genetic aspects -- Congresses.,
  • Fetal growth disorders -- Genetic aspects -- Congresses.,
  • Genetic regulation -- Congresses.,
  • Abnormalities -- genetics -- congresses.,
  • Gene Expression Regulation, Developmental -- congresses.,
  • Fetal Development -- genetics -- congresses.

  • Edition Notes

    Includes bibliographical references and index.

    Statementeditors, Maria Enrica Martini-Neri, Giovanni Neri, John M. Opitz.
    SeriesBirth defects, original article series / March of Dimes Birth Defects Foundation ;, v. 30, no. 1, Birth defects original article series ;, v. 30, no. 1.
    ContributionsMartini-Neri, Maria Enrica., Neri, Giovanni., Opitz, John M., March of Dimes Birth Defects Foundation.
    Classifications
    LC ClassificationsRG626 .B63 v. 30, no. 1, RG627 .B63 v. 30, no. 1
    The Physical Object
    Paginationxxii, 438 p. :
    Number of Pages438
    ID Numbers
    Open LibraryOL974515M
    ISBN 10047113791X
    LC Control Number96011482

    Epigenetic processes prevent female fetuses from becoming masculinized when testosterone exposure gets too high, and vice versa for males. Multiple approaches, including promoter analyses in cultured cells, in adult heart and in transgenic mice, are required to dissect the activity in time and space of cardiac regulatory genes and their combinatorial interactions. Trends Genet ; — Note: Content may be edited for style and length. This fish occurs in two forms: 1 populations that inhabit deep, open water and have a spiny pelvic fin that deters larger predator fish from feeding on them; 2 populations from shallow water environments and lack this spiny pelvic fin.

    Their bodies are patterned by a system of switching which causes development of different features to begin earlier or later, to occur in this or that part of the embryo, and to continue for more or less time. Contrary to expectation, methylation does not necessarily mean silencing; instead, the effect of methylation depends upon the default state of the region. Eberhart, Scott E. Pseudogenes have mutations that prevent them from being expressed at all. CrossRef Google Scholar

    Recruitment of the tinman homolog Nkx J Cell Biol ; — It was evident that the gene must be ancient, dating back to the last common ancestor of bilateral animals before the Ediacaran Period, which began some million years ago. Molecular cloning of rat cardiac troponin I and analysis of troponin I isoform expression in developing heart. References 1.


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Gene regulation and fetal development book

Gene expression in the developing embryo and fetus.

It took a century before these ideas were shown to be correct. Free shipping for individuals worldwide Usually dispatched within 3 to 5 business days. Gene expression in the developing embryo and fetus. Gene duplication events can occur through rare errors in processes such as DNA replication, meiosis, or transposition.

They only rarely reduce the fitness of offspring. They may even contribute to the evolution of new species. Isoform transitions of the myosin-binding protein C family in developing human and mouse muscles.

Mutants lacking this element, and therefore the large pelvic fin righthave been selected for in shallow-water environments. Cardiac myosin binding protein C gene is specifically expressed in heart during murine and human development.

Biologists including Ernst Mayr had believed that eyes had arisen in the animal kingdom at least 40 times, as the anatomy of different types of eye varies widely. This text contains additional chapters on the embryo, placenta, and parturition, to insure that this is a fully self-contained introduction.

It must be possible to erase and re-establish imprints through each generation so that genes that are imprinted in an adult may still be expressed in that adult's offspring.

Regulation of cardiac gene expression during development. PubMed Google Scholar 3. Moreover, epi-marks are usually produced from scratch with each generation — but new evidence is showing that they can sometimes carryover from parent to child.

More recently, genetic screens in animal models have provided additional insight into the genetics of FASD. Mutations can occur in either the regulatory regions e. In addition, qualitative and quantitative changes in gene expression that deviate from the norm may provide a potential marker system for predicting future developmental defects, a system that would be particularly useful in preimplantation embryo technologies before recipient transfer.

PubMed Google Scholar 7. It was a simple, clear and nearly comprehensive picture: but it did not explain embryology. Advertisement Normally, sex-specific marks that are triggered during early fetal development work to protect boys and girls in the womb from undergoing too much natural variation in testosterone, which should normally happen later in a pregnancy.

Uncovering the genetics behind fetal alcohol spectrum disorders

Circ Res ; — Imprinting of whole chromosomes has been reported in mealybugs Genus: Pseudococcus. These analyses prompted searches for susceptibility loci involved in ethanol metabolism and analyses of candidate loci, based on phenotypes observed in FASD.

A hormone-encoding gene identifies a pathway for cardiac but not skeletal muscle gene transcription. PubMed Google Scholar When these regulatory elements control the imprinting of one or more genes, they are known as imprinting control regions ICR.This paradoxical pattern calls for an explanation.

We use published data on fetal androgen signaling and gene regulation via nongenetic changes in DNA packaging (epigenetics) to develop a new model for homosexuality. It is well established that fetal androgen signaling strongly influences sexual atlasbowling.com by: Boggaram V, Margana RK.

Developmental and hormonal regulation of surfactant protein C (SP-C) gene expression in fetal lung: role of transcription and mRNA stability. J Biol Cited by: 3. This is a topical volume consisting of the latest results in cellular, molecular, chromosomal, morphological and clinical studies regarding gene regulation and foetal development.

Gene context of Fetal Development. Furthermore, they mark the emergence of satellite cells during fetal development, and do not require Pax3 function. These results show that Igf2r is paternally imprinted and reveal that the receptor is crucial for regulating normal fetal.

Jun 24,  · Figure Development of a large, spiny pelvic fin in deep-water stickleback (left) depends on the presence of a particular enhancer element upstream of a gene called Pitx.

Evolutionary developmental biology

Mutants lacking this element, and therefore the large pelvic fin (right), have been selected for in shallow-water environments. (Wikipedia-Richard Wheeler-GFDL). The imperfect mouse model / Tom Strachan and Susan Lindsay --The Carnegie Staging of human embryos: a practical guide / Philip Bullen and David Wilson --The ethics of human embryo studies / Rebecca Bennett and John Harris --Establishing new human embryo collections for documenting gene expression in early human development / Philip Bullen.